Judith Goldfinger. Takotsubo cardiomyopathy is classically stress induced and characterized by regional wall motion abnormalities in the absence of coronary occlusion. It predominantly affects postmenopausal women; emotional and physical stressors can trigger the classic cardiomyopathic findings. These changes are likely mediated by
In takotsubo cardiomyopathy (also called transient apical ballooning and stress cardiomyopathy), left ventricular dysfunction, which can be remarkably depressed, recovers within a few weeks.1–4
Takotsubo cardiomyopathy (or broken heart syndrome) is a sudden, temporary heart condition that can cause symptoms like those of a heart attack, such as chest pain and shortness of breath. An electrocardiogram (EKG) may even confirm signs of heart attack. However, an angiogram (X-ray of the blood vessels) shows no blockage or constriction, like Introduction. Takotsubo Syndrome (TTS) – also known as Takotsubo Cardiomyopathy, stress-induced cardiomyopathy, transient apical ballooning and broken heart syndrome – is a condition that is generally triggered by emotional or physical stress and characterised by transient regional left ventricular (LV) systolic dysfunction. Reverse takotsubo, a variant form of takotsubo cardiomyopathy in which the basal and midventricular segments of the left ventricle are akinetic, occurs in a minority of patients [ 1 ]. The majority of takotsubo cardiomyopathy patients recover cardiac function within three to six months. We present the case of two patients who had reversibleIs an important differential diagnosis in patients with acute chest pain Takotsubo cardiomyopathy (also called stress induced cardiomyopathy, apical ballooning, or broken heart syndrome) was first described in Japan 20 years ago.1 It is characterised by acute, reversible left ventricular dysfunction in a characteristic distribution, which does not correlate with the epicardial coronary arteryEEpfS. 375 0 26 143 326 47 39 89 237